Cholesterol-lowering drug, Fenofibrate, could see coronavirus treated like common cold, study finds

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Coronavirus Outbreak

The Muslim Times has the best collection of articles for the war against Covid 19, especially the vaccines.

Source: Yahoo and The Telegraph

By Mason Boycott-Owen

A cholesterol-lowering drug could make coronavirus as treatable as the common cold, scientists have suggested. Researchers at New York’s Mount Sinai Medical Center looked at depriving the virus of nutrients which Covid-19 needs to survive. They found that fat which accumulates inside lung cells is a key component of what the virus needs to reproduce. Depriving the virus of these conditions could mean that the virus could be better controlled, with the researchers claiming it could be reduced to something akin to an ordinary cold. “By understanding how the SARS-CoV-2 controls our metabolism, we can wrestle back control from the virus and deprive it from the very resources it needs to survive,” said Prof Yaakov Nahmias from the Hebrew University of Jerusalem. He continued: “With second-wave infections spiking in countries across the globe, these findings couldn’t come at a better time. “If our findings are borne out by clinical studies, this course of treatment could potentially downgrade Covid-19’s severity into nothing worse than a common cold.” During the study, which was previewed by the Cell Press – a publisher of biomedical journals, including Cell and Neuron – scientists screened medications that could interfere with the virus’s ability to reproduce. They found that one cholesterol-lowering drug, fenofibrate, showed promising results which allowed lung cells to burn more fat and therefore depriving the coronavirus of the conditions it needed to survive. After five days of treatment with the drug, the researchers said that the virus had almost completely disappeared in lab studies.

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    A study conducted by professor Yaakov Nahmias at Hebrew University in Israel has found that an existing cholesterol drug, fenofibrate, could ‘downgrade’ Covid-19 threat level to that of a common cold.

    The findings allegedly come from lab tests on human lung tissue infected with SARS-CoV-2, the virus that causes Covid-19.

    According to the research, the virus leads to deposits of lipids in the lungs. Nahmias partnered with Mount Sinai Medical Center researcher Dr Benjamin tenOever to gain better insights into SARS-CoV-2 mechanism of attack on the human body.

    The researchers observed that the virus changes lipid metabolism in human lungs. They believe that halting this process could help prevent the onset of problems that increase the severity of the disease.

    While SARS-CoV-2 hinders the ability of the body to break down fat, fenofibrate starts this process by binding and activating the DNA site that is blocked by the virus.

    Nahmias was quoted by The Times of Israel as saying: “The interesting thing about our study is that fenofibrate actually binds and activates the very site on the DNA that the virus shuts down — a part of our DNA that allows our cells to burn fat.

    “Virus infection causes the lung cells to start building up fat, and fenofibrate allows the cells to burn it.”

    https://www.pharmaceutical-technology.com/news/study-fenofibrate-covid-19/

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